Opened 9 years ago
Closed 9 years ago
#951 closed enhancement (fixed)
Unquoted numbers in last field of a record break BibTeX import
| Reported by: | dstillman | Owned by: | simon |
|---|---|---|---|
| Priority: | major | Milestone: | 1.0.4 |
| Component: | ingester | Version: | 1.0 |
| Keywords: | Cc: |
Description
Numbers without quotation marks or curly braces that are the last field of a record end a BibTeX import with the already-imported items left behind and no indication that the import was interrupted.
Change History (2)
comment:1 Changed 9 years ago by simon
comment:2 Changed 9 years ago by simon
- Resolution set to fixed
- Status changed from new to closed
(In [2462]) - remove extraneous HTTP request from Time translator when handling single pages
- closes #943, processDocuments broken by Firefox 3 (should fix the specific issue the Time translator was having, if it's seen in other translators. reopen this ticket if there are other problems.)
- closes #649, Importing large BibTeX file fails
- closes #687, Ignore text outside entries in BibTeX files
- closes #951, Unquoted numbers in last field of record break BibTeX import
- closes #932, Support @STRING variables in BibTeX
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Do we have a broken file for this? I created a file with the following entry in the middle, but it worked fine.
@article{bielsky_profound_2004, title = {Profound impairment in social recognition and reduction in anxiety-like behavior in vasopressin V1a receptor knockout mice}, issn = {0893133}, abstract = {Considerable evidence suggests that arginine vasopressin (AVP) is critically involved in the regulation of many social and nonsocial behaviors, including emotionality. The existence of two AVP receptors in the brain, namely the V1a and V1b subtypes, and the lack of clear pharmacological data using selective agonists or antagonists, make it difficult to determine which receptor is responsible for the AVP-mediated effects on behavior. Here we report the behavioral effects of a null mutation in the V1a receptor (V1aR) in male mice. Male mice lacking functional V1aR (V1aRKO) exhibit markedly reduced anxiety-like behavior and a profound impairment in social recognition. V1aRKO performed normally on spatial and nonsocial olfactory learning and memory tasks. Acute central administration of AVP robustly stimulated stereotypical scratching and autogrooming in wild-type (WT), but not V1aRKO males. AVP and oxytocin (OT) mRNA and OT receptor-binding levels were similar in WT and V1aRKO mice. Given the current findings, the V1aR may provide a novel potential pharmacological target for social and affective disorders including autism, and anxiety disorders.}, journal = {Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology}, author = {Isadora F Bielsky and Shuang-Bao Hu and Kathleen L Szegda and Heiner Westphal and Larry J Young}, note = {PMID: 14647484}, keywords = {Animals,Anxiety,Female,Genotype,Male,Maze Learning,Mice,Mice, Inbred C57BL,Mice, Knockout,Receptors, Vasopressin,Recognition (Psychology),Social Behavior}, pages = {483-93}, volume = 29 }